by caryn (10177 Posts), Sat May 08, 2010 04:58 pm
Nicotine significantly facilitated endothelial migration and tube formation. By contrast, soluble fms-like tyrosine kinase 1 and/or soluble endoglin suppressed these endothelial functions. Nicotine restored these soluble fms-like tyrosine kinase 1 and/or soluble endoglin-reduced endothelial functions. Placental growth factor, but not transforming growth factor-beta1, production was significantly stimulated by the presence of nicotine...
Well, that's probably why smokers have a lower incidence. I wonder if that's why we invented smoking. (DO NOT START SMOKING. Yes, it is incredibly frustrating that doing everything right makes it more likely that you'll get preeclampsia, but drugs and cigarettes are still really, really bad for you.)
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