"...the levels of sFlt-1/PlGF ratio and sEng in women with GH were lower than in those with h-PE and with si-PE; however, the majority of women with GH showed abnormal increases of both sFlt-1/PlGF ratio and sEng, suggesting that GH may be a subclinical PE in view of serum levels of angiogenesis-related factors..."http://www.ncbi.nlm.nih.gov/pubmed/21048778
So this has been an ongoing debate! Is there such a thing as "just" a little hypertension at the end of pregnancy? Is it even maybe adaptive -- does it lead to overall better outcomes, and how do you decide which outcomes are better, anyway?
This study does at least confirm that women with raised blood pressure at the end of pregnancy do often have higher levels of the soluble factors linked to preeclampsia symptoms than women who do not see those increases in their blood pressure. The same biochemical stuff is going on, just more mildly. That might help explain why the HYPITAT trial
showed a benefit to women with gestational hypertension with induction at 37 weeks, though.