Endothelin a final common pathway in pathophysiology of PE

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Endothelin a final common pathway in pathophysiology of PE

Postby caryn » Tue Jan 24, 2012 03:18 pm

Identification of antiangiogenic, autoimmune, and inflammatory factors produced in response to placental ischemia have provided potential new avenues for future research into novel therapies for the preeclamptic patient, and suggest new therapeutic avenues for the treatment of preeclampsia.

http://www.ncbi.nlm.nih.gov/pubmed/22257796

Just another "oh hey, it's likely to be soluble FLT and soluble endoglin triggering a pissed-off maternal immune system after the placenta runs short of blood" abstract. :D
Science! The articles you don't want to miss:
The Preeclampsia Puzzle (New Yorker) and Silent Struggle: A New Theory of Pregnancy (New York Times)
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Caryn, @carynjrogers, who is not a doctor and who talks about science stuff *way* too much
DS Oscar born by emergent C-section at 34 weeks for fetal indicators, due to severe PE
DD Bridget born by C-section after water broke at 39 weeks after a healthy pregnancy
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