In families with the absence of maternal chronic hypertension (cHTN; n = 70), the index case was 5.9 (95% CI 2.28-16.15; P <0.001) times more likely to be male and most (12/14) affected sibs of male index cases were male, while female index cases had no affected sibs. Our results confirm a male fetal sex preponderance in SPD. In a subgroup analysis of families without cHTN, a significant male bias was found to extend to sibs of index cases.
http://www.ncbi.nlm.nih.gov/pubmed/22407866
This is one that doesn't apply to me, because maternal family history of hypertension was excluded from the sample population. It says the rate of SPD - severe placental dysfunction - is way higher if you're carrying a boy.
